Laboratory of Neurobiophysics
Kagawa School of Pharmaceutical Sciences,Tokushima Bunri University

Laboratory of Neurobiophysics

Laboratory of NeuroBiophysics
Kagawa School of Pharmaceutical Sciences
Tokushima Bunri University
1314-1 Shido, Sanuki, Kagawa 769-2193
JAPAN
Tel: +81-87-899-7477
Fax: +81-87-894-0181

RESEARCH
  • Kishimoto Y , Tsukamoto I, Nishigawa A, Nishimoto A, Kirino Y, Kato Y, Konishi R, Maruyama T, Sakakibara N. (2018) Data on COA-Cl administration to the APP/PS2 double-transgenic mouse model of Alzheimer's disease: improved hippocampus-dependent learning and unchanged spontaneous physical activity. Data in Brief 20, 1877-1883.
  • Connor S.A, Ammendrup-Johnsen I, Kishimoto Y, Tari PK, Cvetkovska V, Harada T, Ojima D, Yamamoto T, Wang Y.T, Craig A.M. (2017) Loss of synapse repressor MDGA1 enhances perisomatic inhibition, confers resistance to network excitation, and impairs cognitive function. Cell Reports 21 (13)
  • Kishimoto Y, Fukumoto K, Nagai M, Mizuguchi A, Kobashi Y (2017) Early contextual fear memory deficits in a double-transgenic amyloid-beta precursor protein/presenilin 2 mouse model of Alzheimer's disease. Int. J. Alz. Dis. 2017, 8584205.
  • Connor S.A*, Ammendrup-Johnsen I*, Chan A.W*, Kishimoto Y*, Murayama C, Kurihara N, Tada A, Ge Y, Lu H, Yan R, LeDue J.M, Matsumoto H, Kiyonari H, Kirino Y, Matsuzaki F, Suzuki T, Murphy T.H, Wang Y.T, Yamamoto T, Craig A.M. (2016) Altered cortical dynamics and cognitive function upon haploinsufficiency of the autism-linked excitatory synaptic suppressor MDGA2. Neuron 91 (5) 1052-1068. (*Co-1st authors)
  • Kishimoto Y, Shishido H, Sawanishi M, Toyota Y, Ueno M, Kubota T, Kirino Y, Tamiya T, Kawai N. (2016) Data on amyloid precursor protein accumulation, spontaneous physical activity, and motor learning after traumatic brain injury in the triple-transgenic mouse model of Alzheimer's disease. Data in Brief